Download Physiology of Excitable Membranes. Proceedings of the 28th by J. Salánki, H. Meves, N. Chalazonitis PDF

By J. Salánki, H. Meves, N. Chalazonitis

Body structure of Excitable Membranes comprises plenary lecture and lots of the papers provided at 5 symposia of the part ""General mobilephone Physiology"" on the twenty eighth overseas Congress of Physiological Sciences.
Organized into forty four chapters, this ebook starts with a dialogue at the ionic mechanisms of excitability of nerve cells. next chapters specialize in cost circulation in nerve membrane; calcium electrogenesis; optical adjustments in the course of electrogenesis; synaptic transmission and modulation; and transmission in autonomic ganglia.

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Extra info for Physiology of Excitable Membranes. Proceedings of the 28th International Congress of Physiological Sciences, Budapest, 1980

Sample text

1 1 + exp{ -z e(V - V )} kT where z1 represents the effective valency of the fast-moving mobile charges, and V is the potential at which they are equally distributed. Table 1 gives the results of four experiments in which the Boltzmann parameters were measured Qi = 50 mV Fig. 3 Plots of Qi and Ti against pulse potential for an axon dialyzed wiîO CsF and bathed in a solution containing 10 mM-trisCl, 11 mM-CaCl2, H O mM-MgCl2, 690 mMsucrose. Holding potential -100 mV. The values of ii at -100 mV are the averages for all the tail records.

COMPONENTS OF THE ASYMMETRY CURRENT IN THE SQUID GIANT AXON R. D. Keynes, G. C. Malachowski, D. F. Van Helden and N. G. Greeff Laboratory of the Marine Biological Association, Plymouth PL1 2PB, UK, and Physiological Laboratory, Cambridge CB2 3EG, UK It is now well established that in the giant axons of squid (Armstrong & Bezanilla, 1974; Keynes & Rojas, 1974) and Myxioola (Bullock & Schauf, 1978), at the no'de of Ranvier in frog nerve (Nonner, Rojas & Stämpfli, 1975), and in various other excitable tissues, an asymmetrical component of the membrane displacement current can be recorded that appears to arise from conformational changes in the sodium gating system.

Fig. 4 shows that the asymmetry current which reappears under these conditions reaches its peak distinctly later than does the current which still persists in the inactivated state, suggesting that the component associated with the actual opening of the sodium channels is somewhat slower than that recorded for subthreshold pulses. There are two types of explanation for this behaviour. On the one hand, there might be two different populations of mobile charges whose properties were similar but not identical, the first set being those involved in the functioning of the sodium channels, and the second set being non-inactivated and not directly implicated in the sodium system.

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