Download Medical and Nutritional Complications of Alcoholism: by Charles S. Lieber PDF

By Charles S. Lieber

In the Western international, alcohol is the main abused drug. famous majority of sufferers with cirrhosis do For the entire consciousness being directed towards heroin, co­ admit to over the top alcohol intake. different tissues caine, and marijuana, the favourite mood-altering drug in can be significantly affected, together with mind, intestine, the us, as in nearly each human society, is middle, endocrine platforms, bone, blood, and muscle. a question usually raised is, "In what means does an alcoholic alcohol. In nature, the fermentation of sugars is the fluctuate from a nonalcoholic?" Inquiries have concerned with significant resource of ethanol, yet how people first encoun­ tered it's doubtful. It probably happened in both mental makeup, behavioral changes, and socio­ fermented fruit juices (wine), fermented grain (beer), or monetary components. extra lately, despite the fact that, actual dif­ fermented honey (mead). no matter if the Paleolithic Stone ferences were delineated. sooner than the improvement Age guy knew of ethanol is undetermined, however it is of varied disorder entities, power ethanol publicity abundantly transparent that his Neolithic descendants have been leads to profound biochemical and morphological acquainted with the made of fermentation. With the alterations. therefore, an alcoholic doesn't reply exception of the unique population of Tierra del Fuego, regularly to alcohol, different medications, or perhaps different poisonous the Australian aborigines, and a few polar tribes, all agents.

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Extra info for Medical and Nutritional Complications of Alcoholism: Mechanisms and Management

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Other factors that playa key role in ethanol metabolism in vivo include availability of cofactors and the capacity of the liver to dispose of their products (NADH and acetaldehyde). , 1964). Another factor that is difficult to assess is blood flow. , 1988). , 1988). , 1988). In general, when unchanged or decreased flow was observed, there was an association with low blood ethanol levels. , 1971). , 1959) and slowing of ethanol metabolism (Larsen, 1971; Krarup and Larsen, 1972). Most studies also fail to mention the time of day that the experiments were carried out.

Biol. Med. 121:681-685, 1966. , and Lieber, C. : High blood alcohol levels in women: Role of decreased gastric alcohol dehydrogenase activity and first pass metabolism. N. Engl. J. Med. 322:95-99, 1990. : Liver alcohol dehydrogenase in a Japanese population. Jpn. J. Legal Med. 26:46-51, 1972. Gale, C. : Neuroendocrine aspects of thermoregulation. Annu. Rev. Physiol. 35:391-430, 1973. Gentry, R. , and Lieber, C. : Chronic alcohol consumption accelerates gastric emptying and increases peak blood ethanol in rats.

33:87-97, 1973. : Activite alcool-d~hydrog~nasique de la muqueuse gastrique sous I'effet de diff~rents traitements ~thanoliques chez Ie rat. Pathol. Bioi. 10:249-253, 1972. , Worner, T. , JuIkunen, R. 1. , and Lieber, C. : Effects of fasting and chronic alcohol consumption on the first pass metabolism of ethanol. Gastroenterology 92: 1169-1173, 1987. , and Lieber, C. : Gastric metabolism of ethanol: implications for its bioavailability in men and women. In: Biomedical and Social Aspects of Alcohol and Alcoholism (K.

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