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By John D. McConnell (auth.), Reginald C. Bruskewitz MD (eds.)

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55. Ruffolo RR, Hieble JP:

Uro/1996, 155:1018-1122. 35. } Uro/1995, 154:1834-1840. 36. Payne C: The breadth of neurology [editoriall. } Uro/1996, 155:1030-1031. Clinical Evaluation of Lower Urinary Tract Symptoms Due to Benign Prostatic Hyperplasia O~--------------------------------~----~--------~------~~------------------~O 23 Medical Management of Benign Prostatic Hyperplasia Timothy O. Moon J 3 With hi t I gi vid n f bnign pr tati hyp rpl i (BPH) in n rl 0% f m nag d 50 Y ar , and prevalen e rate in r a ing with ag , BPH i th mo t common b nign neoplasm of Id rly m n III.

Pharmacokinetic studies have demonstrated tamsulosin to be a long-acting a-blocker with selectivity for the al a-adrenoreceptor [72]. Theoretically, a prostate-specific a-blocker would maximize clinical efficacy while minimizing side effects. Short-term clinical studies to date have shown tamsulosin to increase peak flow rates Atlas of the Prostate O~------------------------------------------------------------------------------~O 32 Randomized, Placebo-Controlled, Double-Blind, Phase III Clinical Trials of Alfuzosin ms..

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