By J. B. West (auth.), T. H. Stanley, R. J. Sperry (eds.)
Anesthesia and the Lung 1992 provides fresh advances within the prognosis, pre-, intra-, and postoperative anesthetic administration of sufferers with lung affliction, providing for pulmonary and non-pulmonary surgical procedure. It additionally bargains with ventilation-perfusion matters, the lung as a metabolic organ, the results of anesthesia on pulmonary mechanics and pulmonary blood stream. additionally, there are chapters that target hypoxia; neighborhood variations within the lung; pulmonary surfactant; fresh advances within the figuring out of pulmonary edema; excessive altitude illness; anesthesia and the keep an eye on of respiring; fresh improvement in oximetry; instrumentation designed to degree pulmonary oxygen stress, pO2 and pCO2 transcutaneously; differential lung air flow; reactive airlines; septic surprise; the grownup respiration misery syndrome and various points of ventilatory aid.
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Extra resources for Anesthesia and the Lung 1992
1992 KilMer Academic Publishers. 54 The material in this chapter is not encyclopedic, but will emphasize mechanisms that are currently thought to be important in a variety of different disease processes. OUTLINE OF CHAPTER A. B. C. D. E. Failure of normal lung defenses Direct injury by inhaled toxins: 02 toxicity as an example Enzyme-mediated destruction of alveolar walls: the concept of "protease-antiprotease imbalance" Injury caused by cellular and humoral mediators of inflammation 1. Inflammatory effector cells 2.
The third force is the tension of the tissue elements in the alveolar wall associated with lung 47 inflation. This may contribute to the circumferential tension of the alveolar wall depending on the geometry. The capillary wall stress can be calculated for typical conditions at failure. 3 microns (6). Gehr et a1. 4 microns. Using these figures, the calculated wall stress is about 8 x 105 dyn/cm2 (8 x 104 N/m 2 ). This is an extremely high stress being approximately the same as the wall stress of the normal aorta which is predominantly composed of collagen and elastin.
Furthermore, the Clara cells may be the chief cells that repopulate the bronchiolar epithelium after injury. They may also differentiate into other specialized epithelial cells. C. Interstitial Cells The basement membranes of the alveolar epithelial and endothelial cells define the boundaries of the alveolar interstitial region. This area, which contains a connective tissue matrix composed of elastin, Types I and III collagen, proteoglycans, and fibronectin, represents about 50% of the tissue volume of the normal lung.